Thorn, Stephanie LGollob, Michael HHarper, Mary-EllenBeanlands, Rob SDekemp, Robert ADaSilva, Jean N2014-10-012014-10-0120132013Thorn et al. EJNMMI Research 2013, 3:48http://www.ejnmmires.com/content/3/1/48http://hdl.handle.net/10393/31668The cardiac PRKAG2 mutation in the γ2-subunit of adenosine monophosphate activated kinase (AMPK) is characterized by excessive glycogen deposition, hypertrophy, frequent arrhythmias, and progressive conduction system disease. We investigated whether myocardial glucose uptake (MGU) was augmented following insulin stimulation in a mouse model of the PRKAG2 cardiac syndrome.enArticle10.1186/2191-219X-3-48